Fwd: NIH/NIMH Intramural Research Program - Postdoctoral Opportunity

Dear Colleagues, Please apply and/or bring this postdoctoral opportunity to the attention of your best students and colleagues. Details are below and in the attachment. All best wishes, Karen ________________________________ /Karen Faith Berman, M.D./ Chief, Clinical & Translational Neuroscience Branch Section on Integrative Neuroimaging Psychosis & Cognitive Studies Section National Institutes of Health, NIMH Intramural Research Program 9000 Rockville Pike, MSC 1365 Building 10, Room 3C103A Bethesda, MD 20892-1365 phone: 301/496-7603 fax: 301/480-7795 _karen.berman(a)nih.gov_ <mailto:karen.berman@nih.gov> *_POSTDOCTORAL FELLOWSHIP IN MULTIMODAL NEUROIMAGING_* *SECTION ON INTEGRATIVE NEUROIMAGING* *CLINICAL & TRANSLATIONAL NEUROSCIENCE BRANCH* *NATIONAL INSTITUTE OF MENTAL HEALTH, NIH * *INTRAMURAL RESEARCH PROGRAM, DHHS, BETHESDA, MD* The Section on Integrative Neuroimaging in the Clinical & Translational Neuroscience Branch of the National Institute of Mental Health Intramural Research Program (NIMH IRP), at the National Institutes of Health (NIH), invites outstanding individuals to apply for a two to five year post-doctoral fellowship at one of the premier research sites in the world. The renowned NIH Clinical Center on the 300 acre Bethesda campus of the NIH, near Washington D.C., houses unsurpassed, state-of-the-art neuroimaging facilities (MRI, PET and MEG) all dedicated to research, as well as superb clinical facilities, and an exciting, interactive research community of hundreds of talented colleagues. The strong scientific environment and outstanding resources at NIH make this a unique opportunity for an innovative scientist. The Branch takes a multidisciplinary approach, with multimodal neuroimaging (sMRI, rMRI, fMRI, DTI, PET, MEG) at its core, but also integrates genetic, neurochemical, neuropsychological, and clinical investigations to study normal human higher cognitive function throughout the lifespan, as well as neuropsychiatric disorders such as Williams syndrome and schizophrenia. The successful candidate will have particular leadership opportunities within our longitudinal study of Williams syndrome; will have access to large, unique, archival datasets; and will help to design new studies. The position is open to (1) recent Ph.D.'s in experimental psychology, cognitive neuroscience, neuroscience, neuropharmacology, or other applicable disciplines; and (2) M.D.'s with training in psychiatry, neurology, nuclear medicine, radiology or other relevant fields. Applicants should have a demonstrated record of excellent scientific writing skills as well as excellent interpersonal and presentation skill. In addition, experience with any of the following will be an advantage: developmental/pediatric neuroimaging, multimodal neuroimaging techniques (MRI, PET, MEG), conducting cognitive neuroscience experiments, and/or neuroimaging of clinical populations. Experience with SPM, FSL, Freesurfer, UNIX/LINUX/ and/or programming skills (MATLAB, C++; Python) is desirable, but not required. The position is open immediately and applications will be accepted until the position is filled. A curriculum vitae, letter of interest outlining experience and research goals, and three letters forwarded directly from recommenders should be sent to: Karen Berman, M.D.; C/O Jasmin B. Czarapata, Ph.D.; NIH Building 10, Rm 3C209; 9000 Rockville Pike; Bethesda MD 20892-1365 USA. (301) 435-7645, or electronically to _jasmins(a)mail.nih.gov_ <mailto:jasmins@mail.nih.gov> DHHS and NIH are Equal Opportunity Employers. A FEW REPRESENTATIVE PUBS… Eisenberg DP, Ianni AM, Wei SM, Kohn P, Kolachana B, Apud J, Weinberger DR, Berman KF: Brainderived neurotrophic factor (BDNF) Val66Met polymorphism differentially affects hippocampal function in medication-free patients with schizophrenia. *Molecular Psychiatry*, 18(6):713-720, 2013. Jabbi M, Kippenhan JS, Kohn P, Marenco S, Mervis CB, Morris CA, Meyer-Lindenberg A, Berman KF: The Williams syndrome chromosome 7q11.23 hemideletion confers hypersocial, anxious personality via altered insula structure and function. *Proceedings of the National Academy of Sciences *Apr 3;109(14):E860-6, 2012. Dreher JC, Kohn P, Kolachana B, Weinberger DR and Berman KF: Variation in dopamine genes influences responsivity of the human reward system. *Proceedings of the National Academy of Science* 106: 617-622, 2009. Meyer-Lindenberg A, Hariri A, Munuz KE, Mervis CB, Mattay VS, Morris CA and Berman KF: Neural correlates of genetically abnormal social cognition in Williams syndrome. *Nature Neuroscience *8:991-993, 2005. Kippenhan JS, Olsen RK, Mervis CB, Morris CA, Kohn PD, Meyer-Lindenberg A and Berman KF: Genetic contributions to human gyrification: Sulcal morphometry in Williams syndrome. *Journal of* *Neuroscience *25:7840-7846, 2005. Buchsbaum BR, Olsen RK, Koch PF and Berman KF: Human dorsal and ventral auditory streams subserve rehearsal-based and echoic processes during verbal working memory. *Neuron *48:687-97, 2005. Meyer-Lindenberg A, Mervis CB, Sarpal D, Koch P, Steele S, Kohn P, Marenco S, Morris CA, Das S, Kippenhan JS, Mattay VS, Weinberger DR and Berman KF: Functional, structural and metabolic abnormalities of the hippocampal function in Williams syndrome. Journal of Clinical Investigation 115:1888-1895, 2005. Meyer-Lindenberg A, Kohn PD, Kolachana B, Kippenhan JS, McInerney-Leo A, Nussbaum R, Weinberger DR, and Berman KF: Midbrain dopamine and prefrontal function in humans: Interaction and modulation by COMT genotype. *Nature Neuroscience *8:594-596, 2005. Meyer-Lindenberg A, Kohn P, Mervis CB, Kippenhan JS, Olsen RK, Morris CA, and Berman KF: Neural basis of genetically determined visuospatial construction deficit in Williams syndrome. *Neuron* 43:623-631, 2004. (accompanying commentary: “Fulfilling the Promise of the Cognitive Neurosciences,” *Neuron *43:595-596, 2004) Meyer-Lindenberg A, Miletich RS, Kohn PD, Esposito G, Carson RE, Quarantelli M, Weinberger DR and Berman KF: Reduced prefrontal activity predicts exaggerated striatal dopaminergic function in schizophrenia. *Nature Neuroscience *5:267-271, 2002.